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Botensilimab activates current T cells, eliminates regulatory T cells, primes and expands new T cells, and establishes memory cells for durable immunity. Botensilimab is the main CTLA-4 inhibitor to display clinical responses throughout nine cold and therapy-resistant cancers.
AGEN2373 has shown preliminary clinical action and has become very well tolerated by patients without indications of liver toxicity.
Fc-Increased anti-TIGIT bispecific which targets a 2nd main inhibitory receptor expressed on T and NK cells to further improve anti-tumor action
Agenus is building balstilimab as a backbone agent for blend trials inside of its portfolio, along with supplying drug to collaborators to enable novel combos with exterior agents.
Conditionally active antibody intended to activate T and NK cells while mitigating liver toxicities common for the CD137 focus on class
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Botensilimab activates existing T cells, gets rid of regulatory T cells, primes and expands new T cells, and establishes memory cells for long lasting immunity. Botensilimab is the first CTLA-4 inhibitor to show scientific responses across 9 cold and procedure-resistant cancers.
Scientific trials have proven its efficacy in various indications, including a Stage 2 study in cervical cancer wherever it shown strong activity in combination with balstilimab.
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AGEN2373 binds to a unique epitope made to accomplish this response exclusively throughout the tumor microenvironment. This selective binding is meant to steer clear of severe Uncomfortable side effects affiliated with CD137 activation during the liver that were described by Agen8 competitor molecules.
Agenus is building balstilimab to be a backbone agent for mixture trials in just its portfolio, and also providing drug to collaborators to enable novel mixtures with exterior agents.